Adamax commonly refers to an N-acetylated Semax variant sold in research catalogs. Acetylation alters N-terminal charge and may affect stability and permeability in model systems — it is a distinct chemical entity from unmodified Semax.
Dihexa is a synthetic oligopeptide developed as a small-molecule mimetic of hepatocyte growth factor signaling. Preclinical literature emphasizes synaptogenesis and cognitive endpoints in rodent models — with no established human clinical program for catalog material.
N-acetyl Selank amidate applies N-terminal acetylation and C-terminal amidation to the Selank heptapeptide sequence. Catalog listings treat it as a stabilized Selank derivative, but peer-reviewed literature on this exact modified sequence is thinner than for parent Selank.
N-acetyl Semax amidate combines N-terminal acetylation with C-terminal amidation on the Semax heptapeptide backbone. Both modifications are common medicinal-chemistry strategies for peptide stability — the result is a chemically distinct research material from Semax and Adamax.
P21 is a synthetic peptide derived from ciliary neurotrophic factor (CNTF) sequence logic, studied in preclinical models of neurogenesis and cognitive recovery after brain injury. Catalog naming overlaps with unrelated cell-cycle terminology — sequence confirmation is essential.
Selank is a synthetic heptapeptide derived from tuftsin with an added Gly-Pro C-terminal extension. Russian and Western literature describes anxiolytic-like effects in rodent models without classical benzodiazepine sedation, plus immunomodulatory research angles.
Semax is a synthetic heptapeptide analog of the ACTH(4-10) fragment developed in Russian neuropharmacology programs. Western-indexed literature explores neurotrophic gene expression, BDNF-related signaling, and neuroprotection in rodent models.
Community content — not medical advice. Research use only; nothing here is instruction for human use.
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